The gallon jugs were shipped to store locations in six states nationwide

By Moná Thomas 

Published on January 15, 2026 11:55AM EST

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NEED TO KNOW

• 38,043 gallons of Meijer Steam Distilled Water have been recalled
• The enforcement reports cite “floating black foreign substance” contamination for the recall
• The company has yet to issue a statement confirming the nature of the foreign substance

More than 38,000 gallons of bottled water have been recalled after an Enforcement Report from the U.S. Food and Drug Administration (FDA) revealed a “floating black foreign substance” appearing inside gallon-sized jugs.

According to a notice published by the FDA, the recall involves Meijer Steam Distilled Water, which is sold in one-gallon plastic containers with red caps. Meijer voluntarily initiated the recall in November 2025, and it remains ongoing as officials continue to review the issue. In total, 38,043 gallons of the product are affected.

The affected jugs can be identified by a best-by date of Oct. 4, 2026, along with lot code 39-222 #3 and a UPC code of 041250841197. Meijer item codes tied to the recall include Product ID 472859 and Item Code 477910.

The recalled water was distributed to Meijer stores across Illinois, Indiana, Kentucky, Michigan, Ohio and Wisconsin. Consumers who purchased distilled water in those states are urged to check their containers carefully.

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According to the FDA notice, the issue stems from the presence of a black substance floating inside the water, though the exact source and composition of the material have not been publicly identified. The agency has not yet assigned a recall classification, which typically indicates how serious a potential health risk may be.

Meijer did not initially respond to PEOPLE’s request for comment.

Distilled water is often used for more than just drinking. Many consumers rely on it for medical devices, such as CPAP machines, according to Verywell Health, as well as for infant formula preparation and sinus rinses, where water purity is especially important. Because of that, officials say consumers should stop using the recalled water immediately, even if no health issues are apparent.

At this time, no illnesses or injuries have been reported in connection with the recalled product. Still, the FDA advises anyone who has the affected water to either dispose of it safely or return it to a Meijer store for a refund or replacement.

The FDA continues to monitor the recall and has not provided additional details about how the contamination occurred. Consumers are encouraged to review any distilled water they have on hand and follow recall guidance to avoid potential exposure.

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https://people.com/over-38-000-gallons-of-water-have-been-recalled-due-to-foreign-black-substance-contamination-11885997?

• Maya Spaur, 
• Danielle N. Medgyesi, 
• Komal Bangia, 
• Jessica M. Madrigal, 
• Lauren M. Hurwitz, 
• Laura E. Beane Freeman, 
• Jared A. Fisher, 
• Emma S. Spielfogel, 
• James V. Lacey Jr., 
• Tiffany Sanchez, 
• Rena R. Jones & 
• Mary H. Ward 

Journal of Exposure Science & Environmental Epidemiology volume 35, pages454–465 (2025)Cite this article

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Abstract
Background
Pollutants including metals/metalloids, nitrate, disinfection byproducts, and volatile organic compounds contaminate federally regulated community water systems (CWS) and unregulated domestic wells across the United States. Exposures and associated health effects, particularly at levels below regulatory limits, are understudied.

Objective
We described drinking water sources and exposures for the California Teachers Study (CTS), a prospective cohort of female California teachers and administrators.

Methods
Participants’ geocoded addresses at enrollment (1995–1996) were linked to CWS service area boundaries and monitoring data (N = 115,206, 92%); we computed average (1990–2015) concentrations of arsenic, uranium, nitrate, gross alpha (GA), five haloacetic acids (HAA5), total trihalomethanes (TTHM), trichloroethylene (TCE), and tetrachloroethylene (PCE). We used generalized linear regression to estimate geometric mean ratios of CWS exposures across demographic subgroups and neighborhood characteristics. Self-reported drinking water source and consumption at follow-up (2017–2019) were also described.

Results
Medians (interquartile ranges) of average concentrations of all contaminants were below regulatory limits: arsenic: 1.03 (0.54,1.71) µg/L, uranium: 3.48 (1.01,6.18) µg/L, GA: 2.21 (1.32,3.67) pCi/L, nitrate: 0.54 (0.20,1.97) mg/L, HAA5: 8.67 (2.98,14.70) µg/L, and TTHM: 12.86 (4.58,21.95) µg/L. Among those who lived within a CWS boundary and self-reported drinking water information (2017–2019), approximately 74% self-reported their water source as municipal, 15% bottled, 2% private well, 4% other, and 5% did not know/missing. Spatially linked water source was largely consistent with self-reported source at follow-up (2017–2019). Relative to non-Hispanic white participants, average arsenic, uranium, GA, and nitrate concentrations were higher for Black, Hispanic and Native American participants. Relative to participants living in census block groups in the lowest socioeconomic status (SES) quartile, participants in higher SES quartiles had lower arsenic/uranium/GA/nitrate, and higher HAA5/TTHM. Non-metropolitan participants had higher arsenic/uranium/nitrate, and metropolitan participants had higher HAA5/TTHM.

Impact
Though average water contaminant levels were mostly below regulatory limits in this large cohort of California women, we observed heterogeneity in exposures across sociodemographic subgroups and neighborhood characteristics. These data will be used to support future assessments of drinking water exposures and disease risk.

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Introduction
Drinking water represents an important source of exposure to inorganics (e.g., arsenic and nitrate), radionuclides (uranium, alpha particles), disinfection byproducts (DBPs), and volatile organic compounds (VOCs) for populations in the United States (U.S.) and worldwide [1]. Approximately 90% of the U.S. population is served by public water systems, and 10% by private wells [2]. In California, approximately 95% of the population is served by public water systems [3]. Public water systems include at least 15 service connections or serve at least 25 people; community water systems (CWS) are a type of public water system that serve the same population year-round [2]. Public water systems are regulated by the U.S. Environmental Protection Agency (EPA) under the Safe Drinking Water Act [4]. The contaminants we evaluated are regulated through federally enforceable maximum contaminant levels (MCLs), which were determined based on economic and technical feasibility, treatment technologies, cost-benefit analysis, and public health benefit for specific health endpoints [4]. States generally have primacy over enforcement of federal drinking water regulations. Notably, the MCL goal, a non-enforceable standard based solely on risk to health, is 0 µg/l for arsenic, uranium, alpha particles, trichloroethylene (TCE), tetrachloroethylene (PCE), bromodichloromethane, bromoform, and dichloroacetic acid, as there is no known safe level of exposure to these contaminants [4]. Private wells are not federally regulated or monitored.

Nitrate is a common contaminant of drinking water supplies in agricultural areas, due to use of nitrogen fertilizers and concentrated animal feeding operation waste [5, 6]. Atmospheric deposition, erosion of natural deposits, and septic tank or sewage leakage contribute to nitrate contamination in rural and urban areas [4]. Geogenic arsenic occurs in groundwater across the U.S., with regional differences due to climatic and geological factors; arid climates can cause evaporative concentration of arsenic in shallow groundwater supplies and lead to high levels, such as in the San Joaquin Valley of California [7,8,9,10]. Mining and historical arsenical pesticide use are anthropogenic sources of arsenic contamination in water supplies [8]. Uranium is present in different rock types and is leached from host mineral phases to surface and ground water supplies; uranium mining/milling and mobilization of uranium via nitrate fertilizer use are anthropogenic sources of uranium contamination [11,12,13,14,15]. Uranium and other radionuclides can decay and release alpha radiation, often quantified as total gross alpha for monitoring compliance purposes. DBPs are formed by the reaction of chlorine and bromine with natural organic compounds during the disinfection of water supplies to treat pathogens [16]. DBPs are commonly found in public water supplies across the U.S., with the highest concentrations observed in those reliant on surface water or shallow groundwater [16]. While over 700 DBPs have been identified, the most abundant classes are trihalomethanes (THMs, which include the chemicals chloroform, dibromochloromethane, bromodichloromethane, and bromoform, and regulated as the sum total, TTHM), and haloacetic acids (HAA5, regulated as the sum of dichloroacetic acid, trichloroacetic acid, monochloroacetic acid, bromoacetic acid, and dibromoacetic acid) [4, 17, 18]. The VOCs TCE and PCE are solvents used in dry cleaning, metal degreasing, textile, art, and industrial processes, and may be found in some consumer products [19]. Toxic waste disposal sites, sometimes recognized as Superfund sites under the Comprehensive Environmental Response, Compensation, and Liability Act (CERCLA), are anthropogenic sources of inorganic arsenic, uranium, TCE, and PCE in groundwater [12, 19,20,21,22,23,24].

Numerous studies implicate one or more of these drinking water contaminants in adverse health effects, including cancer, cardiovascular disease, reproductive and developmental toxicity, nephrotoxicity, and other adverse health conditions [1, 16, 20, 25,26,27,28,29,30,31,32,33,34,35,36,37,38,39]. Inorganic arsenic is classified by the International Agency for Research on Cancer (IARC) as a cause of cancers of the bladder, lung, and skin, and is associated with increased risk of cancers of the kidney, liver, and prostate [26]. Inorganic arsenic is also a potent toxicant associated with numerous adverse health outcomes, including cardiovascular disease, hypertension, and reproductive disorders [26, 31, 32]. Uranium exposure through drinking water is associated with renal damage and nephrotoxicity, and an increased risk of colorectal, breast, kidney, prostate, and total cancer [20, 27, 33]. Nitrate is classified by IARC as a probable human carcinogen when ingested under conditions that result in the endogenous formation of N-nitroso-compounds, most of which are animal carcinogens [28, 34]. Cancers of the stomach, colon, bladder, kidney, ovary, and thyroid, and thyroid disease are associated with elevated nitrate ingested from drinking water; however, the number of studies of most cancer sites is limited [29, 35]. Higher intake of DBPs through drinking water is associated with increased bladder cancer risk, and a limited number of studies suggest DBP exposures are potential risk factors for colon, rectum, and endometrial cancer [16, 36]. TCE is classified as carcinogenic to humans based on kidney cancer, and PCE (Group 2A) as probably carcinogenic to humans based on bladder cancer evidence [30]. Occupational studies also support adverse developmental, neurological, and hepatotoxic effects of TCE and PCE exposures [19]. Assessment of long-term drinking water contaminant exposures and associated health risks have traditionally been limited by the lack of water quality data that could be assigned to individuals in epidemiologic cohorts; understanding large-scale water quality data at the level of consumer intake is a critical research gap [40]. Additionally, there are relatively few cohort studies evaluating drinking water exposures at levels below the MCLs and World Health Organization guidelines that are commonly experienced by the general U.S. population [1]. Inequalities in CWS arsenic, uranium, and nitrate exposures by sociodemographic characteristics such as, race and ethnicity, income, education, region, and rurality/urbanicity have been documented [41, 42]. Few studies have evaluated sociodemographic inequalities in DBP and TCE/PCE exposures in the United States.

Our primary objective for this study was to describe exposure to regulated, frequently detected and measured contaminants in drinking water in the California Teachers Study (CTS), a large prospective cohort of women. We described the spatial linkage of participants’ residences to their drinking water source and corresponding estimates of contaminant concentrations. For a subset, we evaluated the agreement between address-assigned and self-reported drinking water source and described the daily intake of tap water and CWS contaminants. Additionally, we examined inequalities in CWS exposures across sociodemographic groups.

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https://www.nature.com/articles/s41370-024-00703-9

Understanding risk below the current US EPA regulatory standard

Source:Columbia University's Mailman School of Public Health

Summary:Long-term exposure to arsenic in water may increase cardiovascular risk and especially heart disease risk even at exposure levels below the federal regulatory limit, according to new research. A study describes exposure-response relationships at concentrations below the current regulatory limit and substantiates that prolonged exposure to arsenic in water contributes to the development of ischemic heart disease.Share:

FULL STORY

Long-term exposure to arsenic in water may increase cardiovascular disease and especially heart disease risk even at exposure levels below the federal regulatory limit (10µg/L) according to a new study at Columbia University Mailman School of Public Health. This is the first study to describe exposure-response relationships at concentrations below the current regulatory limit and substantiates that prolonged exposure to arsenic in water contributes to the development of ischemic heart disease.

The researchers compared various time windows of exposure, finding that the previous decade of water arsenic exposure up to the time of a cardiovascular disease event contributed the greatest risk. The findings are published in the journal Environmental Health Perspectives.

“Our findings shed light on critical time windows of arsenic exposure that contribute to heart disease and inform the ongoing arsenic risk assessment by the EPA. It further reinforces the importance of considering non-cancer outcomes, and specifically cardiovascular disease, which is the number one cause of death in the U.S. and globally,” said Danielle Medgyesi, a doctoral Fellow in the Department of Environmental Health Sciences at Columbia Mailman School. “This study offers resounding proof of the need for regulatory standards in protecting health and provides evidence in support of reducing the current limit to further eliminate significant risk.”

According to the American Heart Association and other leading health agencies, there is substantial evidence that arsenic exposure increases the risk of cardiovascular disease. This includes evidence of risk at high arsenic levels (>100µg/L) in drinking water. The U.S. Environmental Protection Agency reduced the maximum contaminant level (MCL) for arsenic in community water supplies (CWS) from 50µg/L to 10µg/L beginning in 2006. Even so, drinking water remains an important source of arsenic exposure among CWS users. The natural occurrence of arsenic in groundwater is commonly observed in regions of New England, the upper Midwest, and the West, including California.

To evaluate the relationship between long-term arsenic exposure from CWS and cardiovascular disease, the researchers used statewide healthcare administrative and mortality records collected for the California Teachers Study cohort from enrollment through follow-up (1995-2018), identifying fatal and nonfatal cases of ischemic heart disease and cardiovascular disease. Working closely with collaborators at the California Office of Environmental Health Hazard Assessment (OEHHA), the team gathered water arsenic data from CWS for three decades (1990-2020).

The analysis included 98,250 participants, 6,119 ischemic heart disease cases and 9,936 CVD cases. Excluded were those 85 years of age or older and those with a history of cardiovascular disease at enrollment. Similar to the proportion of California’s population that relies on CWS (over 90 percent), most participants resided in areas served by a CWS (92 percent). Leveraging the extensive years of arsenic data available, the team compared time windows of relatively short-term (3-years) to long-term (10-years to cumulative) average arsenic exposure. The study found decade-long arsenic exposure up to the time of a cardiovascular disease event was associated with the greatest risk, consistent with a study in Chile finding peak mortality of acute myocardial infarction around a decade after a period of very high arsenic exposure. This provides new insights into relevant exposure windows that are critical to the development of ischemic heart disease.

Nearly half (48 percent) of participants were exposed to an average arsenic concentration below California’s non-cancer public health goal <1 µg/L. In comparison to this low-exposure group, those exposed to 1 to <5 µg/L had modestly higher risk of ischemic heart disease, with increases of 5 to 6 percent. Risk jumped to 20 percent among those in the exposure ranges of 5 to <10 µg/L (or one-half to below the current regulatory limit), and more than doubled to 42 percent for those exposed to levels at and above the current EPA limit ≥10µg/L. The relationship was consistently stronger for ischemic heart disease compared to cardiovascular disease, and no evidence of risk for stroke was found, largely consistent with previous research and the conclusions of the current EPA risk assessment.

These results highlight the serious health consequences not only when community water systems do not meet the current EPA standard but also at levels below the current standard. The study found a substantial 20 percent risk at arsenic exposures ranging from 5 to <10 µg/L which affected about 3.2 percent of participants, suggesting that stronger regulations would provide significant benefits to the population. In line with prior research, the study also found higher arsenic concentrations, including concentrations above the current standard, disproportionally affect Hispanic and Latina populations and residents of lower socioeconomic status neighborhoods.

“Our results are novel and encourage a renewed discussion of current policy and regulatory standards,” said Columbia Mailman’s Tiffany Sanchez, senior author. “However, this also implies that much more research is needed to understand the risks associated with arsenic levels that CWS users currently experience. We believe that the data and methods developed in this study can be used to bolster and inform future studies and can be extended to evaluate other drinking water exposures and health outcomes.”

Co-authors are Komal Bangia, Office of Environmental Health Hazard Assessment, Oakland, California; James V. Lacey Jr and Emma S. Spielfogel,California Teacher Study, Beckman Research Institute, City of Hope, Duarte, California; and Jared A Fisher^, Jessica M. Madrigal, Rena R. Jones, and Mary H. Ward^, Division of Cancer Epidemiology and Genetics, National Cancer Institute.

The study was supported by the National Cancer Institute, grants U01-CA199277, P30-CA033572, P30-CA023100, UM1-CA164917, and R01-CA077398; and also funded by the Superfund Hazardous Substance Research and Training Program P42ES033719; NIH National Institute of Environmental Health Sciences P30 Center for Environmental Health and Justice P30ES9089, NIH Kirschstein National Research Service Award Institutional Research Training grant T32ES007322, NIH Predoctoral Individual Fellowship F31ES035306, and the Intramural Research Program of the NCI Z-CP010125-28.

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https://www.sciencedaily.com/releases/2024/10/241023131603.htm